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Myocardial infarction (MI) leads to neutrophils (N) activation. The activation results in extension of the myocardial necrosis mostly due to the toxic superoxide radicals released from the cells.The aim of our study was to estimate the neutrophil capacity to release the superoxide radicals in MI patients. We measured using chemiluminescence (CL) method spontaneous and PMA, fMLP and opsonized zymosan...
Priming of neutrophils leads to an exaggerated oxidase response to agonists such as the peptide formyl-methionyl-leucyl-phenylalanine (fMLP). Primed neutrophils which damage tissue via cytokines and membrane receptors may be implicated in pathogenesis of inflammatory processes. Pentoxifylline (PTX) is a methylxanthine derivate and phosphodiesterase inhibitor. PTX interacts with several types of...
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