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ObjectiveThe chemokine receptor CCR5 may be important for the recruitment of pathogenic T cells to the CNS in multiple sclerosis (MS). We hypothesized that this chemokine receptor might still be important for T‐cell migration during treatment with anti‐very late antigen (VLA)‐4 antibody. We therefore analysed whether natalizumab‐treated MS patients carrying the CCR5 Δ32 deletion allele, which results...
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