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Immunization of H2 p and H2 q congenic C3H mouse strains with the PLP 103-116 peptide elicited two distinct experimental allergic encephalomyelitis (EAE) disease courses. C3H.Q (H2 q ) mice developed an acute-phase disease with classical ascending paralytic signs whereas C3H.NB (H2 p ) developed a highly variable disease course with symptoms originating from CNS above...
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