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Alzheimer's Disease (AD) is complicated by pro-oxidant intraneuronal Fe 2+ elevation as well as extracellular Zn 2+ accumulation within amyloid plaque. We found that the AD β-amyloid protein precursor (APP) possesses ferroxidase activity mediated by a conserved H-ferritin-like active site, which is inhibited specifically by Zn 2+ . Like ceruloplasmin, APP catalytically oxidizes...
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