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Oleoylethanolamide (OEA) is a satiety factor that controls motivational responses to dietary fat. Here we show that alcohol administration causes the release of OEA in rodents, which in turn reduces alcohol consumption by engaging peroxisome proliferator‐activated receptor‐alpha (PPAR‐α). This effect appears to rely on peripheral signaling mechanisms as alcohol self‐administration is unaltered by...
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