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Aims
Acute myeloid leukemia (AML) is an immunophenotypically heterogenous malignant disease. The early immature CD34+ AML cell subpopulation is frequently impervious to intensive chemotherapy, making them largely responsible for relapse of AML. CD34+ AML cells have higher level of Bcl‐2 protein expression than the CD34− subpopulation. As such, development of drugs that specifically target the Bcl‐2...
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