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Introduction: Neutrophil (PMN) functions are regulated by cytosolic calcium ([Ca2+]i). Calcium channel blockade could therefore limit the inflammatory response to shock, but current calcium channel blockers target L-type channels and thus can exacerbate shock. PMN Ca2+ mobilization however, occurs via store-operated calcium channels (SOCC) that can be targeted selectively. We hypothesized that SOCC...
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