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Background
The Philadelphia‐negative chronic myeloproliferative neoplasms (MPNs) are associated with a huge comorbidity burden, including an increased risk of cardiovascular diseases. Recently, chronic inflammation has been suggested to be the driving force for clonal evolution and disease progression in MPN but also potentially having an impact upon the development of accelerated (premature) atherosclerosis...
Gene expression profiling studies have unraveled deregulation of several genes that might be of pathogenetic importance for the development and phenotype of the Philadelphia‐negative chronic myeloproliferative neoplasms. In the context of interferon‐alpha2 as a promising therapeutic agent, we focused upon the transcriptional profiling of interferon‐associated genes in patients with essential thrombocythemia...
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