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Carcinogenesis is often linked to aberrant activation of Wnt/β-Catenin signalling, in many cases caused by activating CTNNB1 mutations (encoding β-Catenin). Recently, β-catenin was established as a decisive regulator of hepatic glutamine synthetase (GS) and cytochrome P450 (CYP) expression in mouse hepatocarcinogenesis. This study was aimed to analyse the connection of β-Catenin signalling and GS/CYP...
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