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Background The endothelial ADP receptor P2Y1 is responsible for a large part of the reactive hyperemia following cardiac ischemia. Tissue plasminogen activator (t-PA) increases during reactive hyperemia. We postulated that the release of t-PA during reactive hyperemia could be mitigated through blocking the coronary endothelial P2Y1 receptor. Methods t-PA was measured in peripheral arterial blood...
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