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Alzheimer’s disease (AD) is characterized by accumulation of amyloid‐beta (Aβ) senile plaques in patients’ brain tissues. Elevated levels of interleukin‐1beta (IL‐1β) have been identified in cerebrospinal fluid of living AD patients and in animal models of AD. Increased expression of IL‐1β and iron accumulation have been identified in microglial cells that cluster around amyloid plaques in AD mouse...
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