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Both the complement system and tissue factor (TF), a key initiating component of coagulation, are activated in sepsis, and cross‐talk occurs between the complement and coagulation systems. C1‐inhibitor (C1‐INH) can act as a regulator in both systems. Our aim in this study was to examine this cross‐talk by investigating the effects of C1‐INH on Escherichia coli‐induced haemostasis and inflammation...
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