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Destructive bone diseases caused by osteolysis are increasing in incidence. They are characterized by an excessive imbalance of osteoclast formation and activation. During osteolysis, the activation of nuclear factor‐κB (NF‐κB) and mitogen‐activated protein kinase (MAPK) signaling pathways are triggered by receptor activator of NF‐κB ligand (RANKL), inflammatory factors, and oxidative stress. Previous...
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