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Les cassures double-brin de l’ADN sont des lésions létales majeures induites par les radiations ionisantes ou par des stress cellulaires endogènes ; elles peuvent être source d’instabilité génétique. Deux mécanismes entrent en compétition pour la réparation des cassures double-brin : la recombinaison homologue et la ligature d’extrémités non homologues (non-homologous end joining, NHEJ). La recombinaison...
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