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Background and Purpose
Transmembrane member 16A (TMEM16A), an intrinsic constituent of the Ca2+‐activated Cl− channel, is involved in vascular smooth muscle cell (VSMC) proliferation and hypertension‐induced cerebrovascular remodelling. However, the functional significance of TMEM16A for apoptosis in basilar artery smooth muscle cells (BASMCs) remains elusive. Here, we investigated whether and how...
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