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The precise mechanism of characteristic Th2 predominance at maternal–fetal interface remains unresolved. In the present study, we investigated roles of the decidua-derived CCL2 in Th2 predominance at maternal–fetal interface. FCM shows that 55% CD56 + CD16 − CD3 − decidual NK, 52% CD4 + T cells and 75% CD14 + monocytes express CCR2. Recombinant human CCL2 (rhCCL2)...
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