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β-arrestin-dependent activation of the ERK MAP kinase downstream of GPCRs typically originates from internalized receptor–β-arrestin–ERK complexes. A new study now reports that β-arrestin 2 is sufficiently ‘primed’ after ‘kissing’ the β1-adrenergic receptor to initiate ERK activation even in the absence of formation of the receptor–β-arrestin–ERK complex and receptor internalization.
Set the date range to filter the displayed results. You can set a starting date, ending date or both. You can enter the dates manually or choose them from the calendar.