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Introduction
Perturbations in the CACNA1C‐encoded L‐type calcium channel α‐subunit have been linked recently to heritable arrhythmia syndromes, including Timothy syndrome, Brugada syndrome, early repolarization syndrome, and long QT syndrome. These heritable arrhythmia syndromes may serve as a pathogenic basis for autopsy‐negative sudden unexplained death in the young (SUDY). However, the contribution...
Heritable arrhythmia syndromes, including Brugada syndrome (BrS) and idiopathic ventricular fibrillation (IVF), may serve as the pathogenic basis for autopsy‐negative sudden unexplained death (SUD) and sudden infant death syndrome (SIDS). Emerging evidence has linked perturbations in the transient outward current (Ito) conducted by the KCND3‐encoded Kv4.3 pore‐forming α‐subunit to BrS or IVF. However,...
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