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Tolerance to experimental autoimmune myasthenia gravis by nasal administration of microgram amounts of acetylcholine receptor (AChR) has been reported. To elucidate the mechanisms behind tolerance induction via the respiratory tract and the involvement of CD4+T cells, we established AChR-specific CD4+CD8−T cell clones from nasally tolerized rats. Nasal tolerance decreased leukocyte function-associated...
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