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Myofibroblasts and their potential to generate angiotensin (Ang) II and transforming growth factor beta 1 (TGF-β1) at sites of infarction in the rat heart have been implicated in tissue repair. These cells likewise contribute to repair in a subcutaneous pouch model of fibrous tissue formation. Their appearance in pouch tissue coincides with high density ACE and Ang II receptor binding, suggesting...
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