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Background: Sudden cardiac death and myocardial infarction have a circadian variation with a peak incidence in the early morning hours. Increased dispersion of repolarization facilitates the development of conduction delay necessary to induce sustained arrhythmia. Both QT‐dispersion and T‐wave peak to T‐wave end (TpTe) have been proposed as markers of dispersion of myocardial repolarization.
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