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Background & Aims
Portal hypertension (PH) can be present in pre‐cirrhotic stages, even in absence of fibrosis in non‐alcoholic steatohepatitis (NASH) patients. Liver endothelial dysfunction (ED) has been shown as responsible for this effect in short‐term dietary animal models. We evaluated the persistence of PH and underlying mechanisms in a long‐term rat model of NASH.
Methods
Sprague‐Dawley...
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