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One hypothesis for the etiology of Parkinson’s disease (PD) is that the formation of proteinaceous inclusion, which is mainly composed of α-synuclein, may contribute to the selective loss of dopaminergic neurons. To further explore the role of α-synuclein in neurodegeneration of PD, we examined the possible effects of aggregated α-synuclein on the intracellular redox state, dopamine level, and cell...
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