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For postmenopausal cardiovascular disease, long‐term estrogen therapy may increase the risk of breast cancer. To reduce this risk, estrogen may be replaced with the phytoestrogen formononetin, but how formononetin acts on vascular endothelial cells (ECs) and breast cancer cells is unclear. Here, we show that low concentrations of formononetin induced proliferation and inhibited apoptosis more strongly...
Without the authors' knowledge, this article was reviewed by fraudulent reviewers. It has since been re‐reviewed and is being replaced by an updated version.
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