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Experimental studies suggest that cardioprotection can be achieved through either the activation of PKC-ɛ prior to the index ischaemic episode or the inhibition of PKC-δ at the onset of reperfusion. However, whether these PKC isoforms exert such divergent roles in human myocardium, subjected to simulated ischaemia–reperfusion injury, is unclear.Human atrial trabeculae were isolated from right atrial...
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