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Vascular cell adhesion molecule-1 a ligand for leukocyte very late activating antigen-4 is a key player in leukocyte extravasation in MS lesions. Natalizumab a monoclonal antibody against VLA-4 blocks this interaction. VCAM-1 and its soluble form are up-regulated during endothelial activation in MS.We investigated the effect of Natalizumab on sVCAM-1 and VLA-4 on circulating leukocytes in MS patients...
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