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Prion diseases are caused by conversion of a normal cell-surface glycoprotein (PrP C ) into a conformationally altered isoform (PrP Sc ) that is infectious in the absence of nucleic acid. Although a great deal has been learned about PrP Sc and its role in prion propagation, much less is known about the physiological function of PrP C . In this review, we will summarize...
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