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Endothelium-dependent vasodilation (EDV), mediated by nitric oxide from nitric oxide synthase (NOS), is impaired after myocardial ischemia-reperfusion. To determine whether abnormal EDV is due to loss of NOS enzyme or to a reduction in NOS activity, correlative studies were performed in dog hearts (n = 15) subjected to 90 minutes ischemia followed by 3.5 hr reflow. Microvascular EDV was assessed...
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