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Nitric oxide (NO) is a potent endothelium-derived relaxing factor which also may modulate cardiomyocyte inotropism and growth via increasing the cGMP level. While both the endothelial nitric oxide synthase (eNOS) and inducible nitric oxide synthase (iNOS) isoforms have been detected in non-human mammalian tissues, expression and localization of the eNOS and iNOS in the normal and failing human heart...
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