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XRCC4 and XLF are key proteins in the repair of DNA double‐strand breaks through nonhomologous end‐joining. Together, they form a complex that stimulates the ligation of double‐strand breaks. Owing to the suggested filamentous nature of this complex, structural studies via X‐ray crystallography have proven difficult. Multiple truncations of the XLF and XRCC4 proteins were cocrystallized, but yielded...
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