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Glutaminyl‐tRNAGln in Helicobacter pylori is formed by an indirect route requiring a noncanonical glutamyl‐tRNA synthetase and a tRNA‐dependent heterotrimeric amidotransferase (AdT) GatCAB. Widespread use of this pathway among prominent human pathogens, and its absence in the mammalian cytoplasm, identify AdT as a target for the development of antimicrobial agents. We present here the inhibitory properties...
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