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Our previous studies and the others have strongly suggested that c-Jun N-terminal kinase (JNK) signaling pathway plays a critical role in ischemic brain injury. Here we reported that Tat-JNK binding domain (JBD) of JNK-interacting protein-1 (JIP-1), a smaller 11-mer peptide corresponding to residues 153–163 of murine JIP-1 conjugated to Tat peptide, perturbed the assembly of JIP-1–JNK3 complexes,...
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