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Brain tissue acidosis is considered to be a contributor to ischemic brain injury. The deleterious effects of marked acidosis may be associated with reperfusion and an excessive entry of Na + into cerebral neurons and glia as intracellular pH is restored by Na + /H + exchange. Normalization of pH, with activation of many calcium-dependent and other phospholipases and proteases...
Elevated levels of the excitotoxic amino acids, glutamate and aspartate, have been implicated in the pathogenesis of neuronal injury and death induced by cerebral ischemia. This study evaluated the contribution of reversed high-affinity, Na + -dependent, glutamate transport to the ischemia-evoked release of glutamate and aspartate using dl-threo-β-benzyloxyaspartate (dl-TBOA), a newly developed...
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