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The adoptive transfer of OVA‐specific Th1 cells into WT mice followed by OVA inhalation induces a significant elevation of airway hyper‐responsiveness (AHR) with neutrophilia but not mucus hypersecretion. Here, we demonstrate that the airway inflammation model, pathogenically characterized as severe asthma, was partly mimicked by i.n. administration of IFN‐γ. The administration of IFN‐γ instead of...
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