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Acute myeloid leukemia (AML) is a hematologic malignancy with increased lethality. We focused on elucidating the role of Neratinib, a tyrosine kinase inhibitor, in the progression of AML and identify the potential mechanisms. Upon the treatment of Neratinib, autophagy suppressor 3‐methyladenine (3‐MA) and ferroptosis stimulator Erastin, the viability and proliferation of HL‐60 cells were evaluated...