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On the basis of the evidence that zinc is released from glutamatergic neuron terminals in the amygdala, the effect of chelation of amygdalar extracellular zinc on glutamate release from the neuron terminals was studied by using in vivo microdialysis. When the amygdala was perfused with 100 μM CaEDTA to chelate extracellular zinc, glutamate concentration in the perfusate was decreased significantly,...
On the basis of the evidence that vesicular zinc may be essential to the functions of the amygdala, the movement and action of actively functioning zinc in synapses in the amygdala of rats were studied using in vivo microdialysis. The increase of 65 Zn release into the amygdalar extracellular space during stimulation with high K + was inhibited by the addition of 1 μM tetrodotoxin...
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