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Among human peripheral blood (PB) monocyte (Mo) subsets, the classical CD14++CD16− (cMo) and intermediate CD14++CD16+ (iMo) Mos are known to activate pathogenic Th17 responses, whereas the impact of nonclassical CD14+CD16++ Mo (nMo) on T‐cell activation has been largely neglected. The aim of this study was to obtain new mechanistic insights on the capacity of Mo subsets from healthy donors (HDs) to...
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