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Myocardial toxic injury is characterized by a drug or chemical interference with a cellular process or structure that results in leakage of subcellular constituents with some loss of cardiomyocyte and cardiac function. Combined biomarkers of cytotoxicity, leakage and dysfunction that translate across cell-based to animal models to humans are needed to predict, detect, quantitate, monitor, understand,...
There is an overwhelming weight of evidence that certifies cardiac troponin (cTn) as the preferred, defacto, translational, safety biomarker for myocardial injury in cardiotoxicity. As well as being the gold standard for cardiac injury in man, it has been widely used for clinical assessment and monitoring of cardiac toxicity in humans being treated for cancer. Furthermore, several dozen preclinical...
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