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Tissue accumulation of the cytotoxic β-amyloid peptide (Aβ) occurs in Alzheimer's disease (AD), one possible source being the platelet. AD and cardiovascular disease may share some risk factors, including hypercholesterolaemia which is associated with increased platelet activity. We examined platelet Aβ release under resting and collagen-stimulated conditions in normocholesterolaemic and hypercholesterolaemic...