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We previously reported that conventional protein kinase C (cPKC)β participated in hypoxic preconditioning‐induced neuroprotection against cerebral ischemic injury, and collapsin response‐mediated protein 2 (CRMP2) was identified as a cPKCβ interacting protein. In this study, we explored the regulation of CRMP2 phosphorylation and proteolysis by cPKCβ, and their role in ischemic injury of oxygen–glucose...
J. Neurochem. (2011) 117, 346–356.
AbstractHypoxic preconditioning (HPC) initiates intracellular signaling pathway to provide protection against subsequent cerebral ischemic injuries, and its mechanism may provide molecular targets for therapy in stroke. According to our study of conventional protein kinase C βII (cPKCβII) activation in HPC, the role of cPKCβII in HPC‐induced neuroprotection and...
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